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Dr. Willmar Schwabe Pharmaceuticals has bought the company Peithner Dr. Wiener Holding GmbH. This acquisition will enable Schwabe increase its presence in Austria and in Central and Eastern Europe (ECE).

Dr. Dirk Reischig, chairman of the board and CEO of Dr. Willmar Schwabe Pharmaceuticals, said: "We are delighted that the group of companies Peithner joins Schwabe.

The behavior of the particle size distribution of coagulating dispersions is studied theoretically. If the collision frequency factor is a homogeneous function of particle volume, the partial integro-differential equation describing the coagulation kinetics can be transformed into an ordinary integro-differential equation by a similarity transformation originally proposed by Friedlander. The solution to the resulting equation, called the self-preserving spectrum, is determined for three different collision mechanisms: (1) constant collision frequency factor, (2) Brownian motion, and (3) simultaneous Brownian motion and shear flow, in which the shear rate decreases with time in a particular way. The results of this study indicate that the shape of the self-preserving spectrum is greatly influenced by the collision mechanism.

An international investigation involving the Consejo Superior de Investigaciones Científicas (CSIC) has characterized a new clinical syndrome with the genetic defect that produces it, allowing an early and accurate diagnosis of a disorder, although known, was not correctly identified. The study, involving researchers from the United States, Israel, Japan and Spain, has important implications in the diagnosis and treatment of patients.

Those affected by the syndrome now described intellectual disabilities and abnormal facial features. It is caused by a disruption of embryonic development in which, according to the results of this study, there is a duplication of the genes are located in the terminal region of the short arm of chromosome 17, linked to the formation of the brain. Researchers have identified two genetic variants (gene duplication of Lis1 and YWHAE) producing two different clinical forms. Despite the identification, the syndrome has not received any special name.